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photoelectric actimeter  (Panlab)

 
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    Panlab photoelectric actimeter
    Photoelectric Actimeter, supplied by Panlab, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/photoelectric+actimeter/actimeter/us12448601-238-11-14
    Average 86 stars, based on 1 article reviews
    photoelectric actimeter - by Bioz Stars, 2026-10
    86/100 stars

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    Related Articles

    Activity Assay:

    Article Title: Acute Colon Inflammation Triggers Primary Motor Cortex Glial Activation, Neuroinflammation, Neuronal Hyperexcitability, and Motor Coordination Deficits
    Article Snippet: .. The spontaneous locomotor activity was measured for 10 min in an active cage, consisting of a novel open-field arena (dimensions 45 × 45 cm) equipped with a photoelectric actimeter (LE8815-Panlab, Barcelona, Spain) with SEDACOM 2.0v software (Barcelona, Spain). ..

    Article Title: Neural tissue unit and use of such a unit for implantation into the nervous system of a mammal
    Article Snippet: .. The “Open Field” Actimeter Spontaneous locomotor activity is measured using a photoelectric actimeter (Actitrack, Panlab, S. L., Barcelona, Spain). ..

    Software:

    Article Title: Acute Colon Inflammation Triggers Primary Motor Cortex Glial Activation, Neuroinflammation, Neuronal Hyperexcitability, and Motor Coordination Deficits
    Article Snippet: .. The spontaneous locomotor activity was measured for 10 min in an active cage, consisting of a novel open-field arena (dimensions 45 × 45 cm) equipped with a photoelectric actimeter (LE8815-Panlab, Barcelona, Spain) with SEDACOM 2.0v software (Barcelona, Spain). ..



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    Molecular hydrogen (H2) inhalation reduces AIMS without interfering with the anti-parkinsonian effect of L-DOPA. (A, B) Schematic protocol. After Sham surgery or 6-OHDA injection, the rats were analyzed in the apomorphine-induced rotational test for lesion confirmation. Then, Sham or 6-OHDA-lesioned rats were treated chronically (15 days) with L-DOPA or its vehicle. Subsequently before the last L-DOPA injection, Air mixture or H2 inhalation was performed for a 1-h period, followed by the L-DOPA injection and abnormal involuntary movements on axial, limb and orofacial parameters (ALO AIMs) and locomotor activity were measured. (C) 6-OHDA lesion significantly increased total contralateral turns induced by apomorphine. (D) Photomicrographs of coronal brain sections illustrating the loss of TH positive immunolabeling in the striatum fibers (D) and substantia nigra compacta neurons (E), scale bars = 1200 μm. (F, G) H2 treatment does not alter the L-DOPA effect on the distance travelled (F) and rearing (G) in the <t>actimeter.</t> (H) Time course of the appearance of dyskinetic manifestation with 6-OHDA + L-DOPA + Air mixture compared to 6-OHDA + L-DOPA + H2 inhalation (p < 0.001) across time (for each 20-min period). (I, J, K) show the co-administration of L-DOPA and H2 inhalation effects on the axial (I), limb (J) and orofacial (K) AIMs over 120 min. Sham rats treated with vehicle of L-DOPA (SHAM + Veh, white); Sham rats treated chronically with L-DOPA (SHAM + L-DOPA, orange); 6-OHDA lesioned rats treated with vehicle of L-DOPA (6-OHDA + Veh, green); 6-OHDA lesioned rats treated chronically with L-DOPA (6-OHDA + L-DOPA, blue). AIMs were analyzed using 2-Way RM ANOVA analysis with Tukey's multiple comparisons post-hoc test. Distance travelled was analyzed by ordinary One-Way ANOVA with Fisher post-hoc test. Data are reported as mean ± SEM. ***p < 0.001, **p < 0.01, *p < 0.05. A total of 42 rats were tested in this experiment (n = 5/6 per group, the specific amounts of each group are indicated in parentheses). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)
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    Molecular hydrogen (H2) inhalation reduces AIMS without interfering with the anti-parkinsonian effect of L-DOPA. (A, B) Schematic protocol. After Sham surgery or 6-OHDA injection, the rats were analyzed in the apomorphine-induced rotational test for lesion confirmation. Then, Sham or 6-OHDA-lesioned rats were treated chronically (15 days) with L-DOPA or its vehicle. Subsequently before the last L-DOPA injection, Air mixture or H2 inhalation was performed for a 1-h period, followed by the L-DOPA injection and abnormal involuntary movements on axial, limb and orofacial parameters (ALO AIMs) and locomotor activity were measured. (C) 6-OHDA lesion significantly increased total contralateral turns induced by apomorphine. (D) Photomicrographs of coronal brain sections illustrating the loss of TH positive immunolabeling in the striatum fibers (D) and substantia nigra compacta neurons (E), scale bars = 1200 μm. (F, G) H2 treatment does not alter the L-DOPA effect on the distance travelled (F) and rearing (G) in the <t>actimeter.</t> (H) Time course of the appearance of dyskinetic manifestation with 6-OHDA + L-DOPA + Air mixture compared to 6-OHDA + L-DOPA + H2 inhalation (p < 0.001) across time (for each 20-min period). (I, J, K) show the co-administration of L-DOPA and H2 inhalation effects on the axial (I), limb (J) and orofacial (K) AIMs over 120 min. Sham rats treated with vehicle of L-DOPA (SHAM + Veh, white); Sham rats treated chronically with L-DOPA (SHAM + L-DOPA, orange); 6-OHDA lesioned rats treated with vehicle of L-DOPA (6-OHDA + Veh, green); 6-OHDA lesioned rats treated chronically with L-DOPA (6-OHDA + L-DOPA, blue). AIMs were analyzed using 2-Way RM ANOVA analysis with Tukey's multiple comparisons post-hoc test. Distance travelled was analyzed by ordinary One-Way ANOVA with Fisher post-hoc test. Data are reported as mean ± SEM. ***p < 0.001, **p < 0.01, *p < 0.05. A total of 42 rats were tested in this experiment (n = 5/6 per group, the specific amounts of each group are indicated in parentheses). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)
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    Molecular hydrogen (H2) inhalation reduces AIMS without interfering with the anti-parkinsonian effect of L-DOPA. (A, B) Schematic protocol. After Sham surgery or 6-OHDA injection, the rats were analyzed in the apomorphine-induced rotational test for lesion confirmation. Then, Sham or 6-OHDA-lesioned rats were treated chronically (15 days) with L-DOPA or its vehicle. Subsequently before the last L-DOPA injection, Air mixture or H2 inhalation was performed for a 1-h period, followed by the L-DOPA injection and abnormal involuntary movements on axial, limb and orofacial parameters (ALO AIMs) and locomotor activity were measured. (C) 6-OHDA lesion significantly increased total contralateral turns induced by apomorphine. (D) Photomicrographs of coronal brain sections illustrating the loss of TH positive immunolabeling in the striatum fibers (D) and substantia nigra compacta neurons (E), scale bars = 1200 μm. (F, G) H2 treatment does not alter the L-DOPA effect on the distance travelled (F) and rearing (G) in the actimeter. (H) Time course of the appearance of dyskinetic manifestation with 6-OHDA + L-DOPA + Air mixture compared to 6-OHDA + L-DOPA + H2 inhalation (p < 0.001) across time (for each 20-min period). (I, J, K) show the co-administration of L-DOPA and H2 inhalation effects on the axial (I), limb (J) and orofacial (K) AIMs over 120 min. Sham rats treated with vehicle of L-DOPA (SHAM + Veh, white); Sham rats treated chronically with L-DOPA (SHAM + L-DOPA, orange); 6-OHDA lesioned rats treated with vehicle of L-DOPA (6-OHDA + Veh, green); 6-OHDA lesioned rats treated chronically with L-DOPA (6-OHDA + L-DOPA, blue). AIMs were analyzed using 2-Way RM ANOVA analysis with Tukey's multiple comparisons post-hoc test. Distance travelled was analyzed by ordinary One-Way ANOVA with Fisher post-hoc test. Data are reported as mean ± SEM. ***p < 0.001, **p < 0.01, *p < 0.05. A total of 42 rats were tested in this experiment (n = 5/6 per group, the specific amounts of each group are indicated in parentheses). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)

    Journal: Brain, Behavior, & Immunity - Health

    Article Title: Effects of hydrogen gas inhalation on L-DOPA-induced dyskinesia

    doi: 10.1016/j.bbih.2023.100623

    Figure Lengend Snippet: Molecular hydrogen (H2) inhalation reduces AIMS without interfering with the anti-parkinsonian effect of L-DOPA. (A, B) Schematic protocol. After Sham surgery or 6-OHDA injection, the rats were analyzed in the apomorphine-induced rotational test for lesion confirmation. Then, Sham or 6-OHDA-lesioned rats were treated chronically (15 days) with L-DOPA or its vehicle. Subsequently before the last L-DOPA injection, Air mixture or H2 inhalation was performed for a 1-h period, followed by the L-DOPA injection and abnormal involuntary movements on axial, limb and orofacial parameters (ALO AIMs) and locomotor activity were measured. (C) 6-OHDA lesion significantly increased total contralateral turns induced by apomorphine. (D) Photomicrographs of coronal brain sections illustrating the loss of TH positive immunolabeling in the striatum fibers (D) and substantia nigra compacta neurons (E), scale bars = 1200 μm. (F, G) H2 treatment does not alter the L-DOPA effect on the distance travelled (F) and rearing (G) in the actimeter. (H) Time course of the appearance of dyskinetic manifestation with 6-OHDA + L-DOPA + Air mixture compared to 6-OHDA + L-DOPA + H2 inhalation (p < 0.001) across time (for each 20-min period). (I, J, K) show the co-administration of L-DOPA and H2 inhalation effects on the axial (I), limb (J) and orofacial (K) AIMs over 120 min. Sham rats treated with vehicle of L-DOPA (SHAM + Veh, white); Sham rats treated chronically with L-DOPA (SHAM + L-DOPA, orange); 6-OHDA lesioned rats treated with vehicle of L-DOPA (6-OHDA + Veh, green); 6-OHDA lesioned rats treated chronically with L-DOPA (6-OHDA + L-DOPA, blue). AIMs were analyzed using 2-Way RM ANOVA analysis with Tukey's multiple comparisons post-hoc test. Distance travelled was analyzed by ordinary One-Way ANOVA with Fisher post-hoc test. Data are reported as mean ± SEM. ***p < 0.001, **p < 0.01, *p < 0.05. A total of 42 rats were tested in this experiment (n = 5/6 per group, the specific amounts of each group are indicated in parentheses). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)

    Article Snippet: Actimeter test To evaluate possible motor effects of the treatments, the spontaneous locomotor activity of each animal was individually assessed in a photoelectric actimeter (Actitrack, Panlab, Barcelona, Spain).

    Techniques: Injection, Activity Assay, Immunolabeling